Alternative splicing occurs in most human genes and contributes to protein diversity by producing multiple m RNAs from each gene. In this book, the authors present new developments in alternative splicing research. Topics discussed include alternative splicing alterations in Alzheimer’s disease; plant RNA-binding proteins implicate m RNA processing in abscisic acid (ABA) responses; comprehensive analyses of alternative exons in neuronally differentiated P19 cells; an epigenetic view on alternative splicing; identification of genuine alternative splicing variants for rare or long-sized transcripts; alternative RNA splicing and regulation of nitric oxide signaling; alternative splicing by analyzing human m RNA diversity using data of FLJ human c DNAs; alternative splicing in human immune systems and autoimmune diseases; and poly (ADP-Ribosyl)ation regulation in alternative splicing.
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